Search results
Results From The WOW.Com Content Network
The specific molecular mechanism that underpins this movement disorder is not well known. [2] However, most researchers suggest that it follows an autosomal dominant genetic inheritance pattern in which mutations in certain genes give rise to structural abnormalities in nervous system networks responsible for voluntary skeletal muscle movement, which, in turn, result in the functional movement ...
Bimanual coordination involves the coordination of two arms in bimanual action, which allows two hands to move simultaneously to do tasks. [1] Examples of bimanual coordination include clapping hands, opening the cap of a bottle with two hands or typing words on a keyboard with both hands.
Facial synkinesis is a common sequela to Idiopathic Facial Nerve Paralysis, also called Bell's Palsy or Facial Palsy. [2] Bell's Palsy, which is thought to occur due to a viral reactivation which can lead (through unknown mechanisms) to diffuse axon demyelination and degeneration of the seventh cranial nerve, results in a hemifacial paralysis due to non-functionality of the nerve.
In medicine, split hand syndrome is a neurological syndrome in which the hand muscles on the side of the thumb (lateral, thenar eminence) appear wasted, whereas the muscles on the side of the little finger (medial, hypothenar eminence) are spared.
The Haken-Kelso-Bunz (HKB) is a theoretical model of motor coordination originally formulated by Hermann Haken, J. A. Scott Kelso and H. Bunz. [1] The model attempts to provide the framework for understanding coordinated behavior in living things.
Eagle syndrome (also termed stylohyoid syndrome, [1] styloid syndrome, [2] stylalgia, [3] styloid-stylohyoid syndrome, [2] or styloid–carotid artery syndrome) [4] is an uncommon condition commonly characterized but not limited to sudden, sharp nerve-like pain in the jaw bone and joint, back of the throat, and base of the tongue, triggered by swallowing, moving the jaw, or turning the neck. [1]
Medications are used to reverse the symptoms of extrapyramidal side effects caused by antipsychotics or other drugs, by either directly or indirectly increasing dopaminergic neurotransmission. The treatment varies by the type of the EPS, but may involve anticholinergic agents such as procyclidine, benztropine, diphenhydramine, and trihexyphenidyl.
Symptoms onset any time from birth to adulthood. [5] The earlier the disease onset, the greater the variety of possible signs and symptoms. [ citation needed ] Thus, various diagnostic classifications based on the age of onset/severity of the disease have been proposed, although DM1 manifestations likely lie on a continuum.