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Download as PDF; Printable version; In other projects Wikidata item; ... This is a list of antineoplastic agents used to treat cancer. Antineoplastic agents INN
Anti-Cancer Agents in Medicinal Chemistry is a peer-reviewed academic journal covering the disciplines of medicinal chemistry and drug design relating to chemotherapeutic agents in cancer. It is published by Bentham Science Publishers and the editor-in-chief is Simone Carradori ("G. d'Annunzio" University of Chieti-Pescara). The journal covers ...
An alkylating antineoplastic agent is an alkylating agent used in cancer treatment that attaches an alkyl group (C n H 2n+1) to DNA. [ 1 ] Since cancer cells, in general, proliferate faster and with less error-correcting than healthy cells, cancer cells are more sensitive to DNA damage—such as being alkylated.
Extracts from Camptotheca (the "happy tree" or "cancer tree") were used to develop the chemotherapeutic drug Topotecan. Plant sources of anti-cancer agents are plants, the derivatives of which have been shown to be usable for the treatment or prevention of cancer in humans.
Cell-cycle nonspecific antineoplastic agents (CCNS) refer to a class of pharmaceuticals that act as antitumor agents at all or any phases of the cell cycle. [ 1 ] Alkylating antineoplastic agent and anthracyclins are two examples.
Cell-cycle nonspecific antineoplastic agents; Chemically linked Fab; Chemoimmunotherapy; Chemoprotective agent; Chemosensitizer; Chemotherapy; Chemotherapy-induced nausea and vomiting; Ciltacabtagene autoleucel; Copanlisib; Cytestrol acetate
Platinum-based antineoplastic drugs (informally called platins) are chemotherapeutic agents used to treat cancer. Their active moieties are coordination complexes of platinum . These drugs are used to treat almost half of people receiving chemotherapy for cancer.
Monomethyl auristatin E is an antimitotic agent which inhibits cell division by blocking the polymerisation of tubulin. The linker to the monoclonal antibody is stable in extracellular fluid, but is cleaved by cathepsin once the conjugate has entered a tumor cell, thus activating the antimitotic mechanism. [3] [4] Structure of a MMAE-MAB-conjugate.