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The oculomotor nerve, also known as the third cranial nerve, cranial nerve III, or simply CN III, is a cranial nerve that enters the orbit through the superior orbital fissure and innervates extraocular muscles that enable most movements of the eye and that raise the eyelid. The nerve also contains fibers that innervate the intrinsic eye ...
Oculomotor nerve palsy or oculomotor neuropathy[1] is an eye condition resulting from damage to the third cranial nerve or a branch thereof. As the name suggests, the oculomotor nerve supplies the majority of the muscles controlling eye movements (four of the six extraocular muscles, excluding only the lateral rectus and superior oblique).
The Edinger–Westphal nucleus (accessory oculomotor nucleus, [1] or visceral oculomotor nucleus [2]) is one of two nuclei of the oculomotor nerve and is located in the midbrain. [1][3] It receives afferents from both pretectal nuclei (which have in turn received afferents from the optic tract). [4] It contains parasympathetic pre-ganglionic ...
The oculomotor nerve (III), trochlear nerve (IV) and abducens nerve (VI) coordinate eye movement. The oculomotor nerve controls all muscles of the eye except for the superior oblique muscle controlled by the trochlear nerve (IV), and the lateral rectus muscle controlled by the abducens nerve (VI). This means the ability of the eye to look down ...
The nucleus of the oculomotor nerve, considered from a physiological standpoint, can be subdivided into several smaller groups of cells, each group controlling a particular muscle. A nearby nucleus, the Edinger-Westphal nucleus lies dorsal to the main oculomotor nucleus. It is responsible for the autonomic functions of the oculomotor nerve ...
The trochlear nerve controls the superior oblique muscle to rotate the eye along its axis in the orbit medially, which is called intorsion, and is a component of focusing the eyes on an object close to the face. The oculomotor nerve controls all the other extraocular muscles, as well as a muscle of the upper eyelid.
Oculomotor apraxia. Oculomotor apraxia (OMA) is the absence or defect of controlled, voluntary, and purposeful eye movement. [1] It was first described in 1952 by the American ophthalmologist David Glendenning Cogan. [2] People with this condition have difficulty moving their eyes horizontally and moving them quickly.
The oculomotor nerve (cranial nerve III) supplies the inferior oblique muscle (along with four other eye muscles – superior rectus, medial rectus, inferior rectus and the striated muscle of levator palpebrae superioris), and when this muscle is non-functional (as in oculomotor palsy) the eye incyclotorts; i.e. twists/rotates inward.