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In cyanopsia, these photoreceptors, particularly the ones sensitive to blue light (short-wavelength cones), are overstimulated or affected, leading to an exaggerated perception of blue hues. Cyanopsia is a symptom rather than a disease, below are some factors that may cause Cyanopsia: [5] Cataract in human eye . Post-Cataract Surgery:
Intraoperative floppy iris syndrome (IFIS) is a complication that may occur during cataract extraction in certain patients. This syndrome is characterized by a flaccid iris which billows in response to ordinary intraocular fluid currents, a propensity for this floppy iris to prolapse towards the area of cataract extraction during surgery, and progressive intraoperative pupil constriction ...
Symptoms may disappear if untreated, but treatment may decrease both the healing time and the chances of remission. [citation needed] Artificial tear eye-drops or ointments may be a suitable treatment for mild cases. Low-dosage steroidal eye-drops, such as prednisone, fluorometholone, loteprednol (Lotemax 0.5%) or rimexolone.
There are many diseases known to cause ocular or visual changes. Diabetes , for example, is the leading cause of new cases of blindness in those aged 20–74, with ocular manifestations such as diabetic retinopathy and macular edema affecting up to 80% of those who have had the disease for 15 years or more.
Tamsulosin, sold under the brand names including Flomax and Contiflo, is a medication used to treat symptomatic benign prostatic hyperplasia (BPH) and chronic prostatitis and to help with the passage of kidney stones. [6] [7] [8] The evidence for benefit with a kidney stone is better when the stone is larger. [8] Tamsulosin is taken by mouth. [6]
It is caused when aqueous humour fails to adequately drain from the eye, resulting in pressure build-up. [10] Keratoconus: vision problems can be caused when the cornea thins and distorts into a conical shape. While the cause is unknown, Keratoconus is believed to be congenital, and can be exacerbated by allergies and eye rubbing. [11]
The diagnosis of toxic or nutritional optic neuropathy is usually established by a detailed medical history and careful eye examination. If the medical history clearly points to a cause, neuroimaging to rule out a compressive or infiltrative lesion is optional. However, if the medical history is atypical or does not clearly point to a cause ...
A proposed explanation for the reduced sensitivity is the induced quiescence of free nerve endings following long term corneal exposure to contact lenses. [23] Long-term use of PMMA or thick hydrogel contact lenses have been found to cause increased eye irritability, photophobia, blurred vision, and persistent haloes. [18]