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Genu varum (also called bow-leggedness, bandiness, ... Treatment for children with Blount's disease is typically braces but surgery may also be necessary.
Knee: genu varum (from Latin genu = knee) — the tibia is turned inward in relation to the femur, resulting in a bowlegged deformity. Ankle: talipes varus (from Latin talus = ankle and pes = foot). A notable subtype is clubfoot or talipes equinovarus, which is where one or both feet are rotated inwards and downwards. [6] [7]
Multiple epiphyseal dysplasia (MED), also known as Fairbank's disease, is a rare genetic disorder (dominant form: 1 in 10,000 births) that affects the growing ends of bones. Long bones normally elongate by expansion of cartilage in the growth plate (epiphyseal plate) near their ends.
Blount's disease is an important differential diagnosis because it causes knee deformities in a similar fashion to rickets namely bow legs or genu varum. Infants with rickets can have bone fractures. This sometimes leads to child abuse allegations.
Temporary hemiepiphysiodesis is widely used to treat angular or coronal plane deformities around the knee in children i.e. deformities occurring in the medial/lateral plane as genu varum/ genu valgum. Additionally, it has been used to treat sagittal plane deformities i.e. deformities arising the anterior/posterior plane.
It is also used to correct a coxa vara, genu valgum, and genu varum. The operation is done under a general anaesthetic. [1] Osteotomy is one method to relieve pain of arthritis, especially of the hip and knee. It is being replaced by joint replacement in the older patient. [2] Due to the serious nature of this procedure, recovery may be extensive.
Osteochondrodysplasias or genetic bone diseases can cause lower extremity deformities similar to Blount's disease. The clinical appearance and the characteristic radiographic are important to confirm the diagnosis. [6] [7] Staples for epiphysiodesis in the bone, Blount's disease treatment - rtg. snapshots and biomechanical experiments [8]
[10] [11] [12] The prevalence of the disease is 1 in 20,000. [13] X-linked hypophosphatemia may be lumped in with autosomal dominant hypophosphatemic rickets under general terms such as hypophosphatemic rickets. Hypophosphatemic rickets are associated with at least nine other genetic mutations. [14]