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Moeller's disease, Cheadle's disease, scorbutus, [1] Barlow's disease, hypoascorbemia, [1] vitamin C deficiency: Scorbutic gums, a symptom of scurvy. The triangle-shaped areas between the teeth show redness of the gums. Specialty: Endocrinology: Symptoms: Weakness, feeling tired, changes to hair, sore arms and legs, gum disease, easy bleeding ...
Rickets, scientific nomenclature: rachitis (from Greek ῥαχίτης rhakhítēs, [6] meaning 'in or of the spine'), is a condition that results in weak or soft bones in children and is caused by either dietary deficiency or genetic causes. [2]
Vitamin C (also known as ascorbic acid and ascorbate) is a water-soluble vitamin found in citrus and other fruits, berries and vegetables. It is also a generic prescription medication and in some countries is sold as a non-prescription dietary supplement. As a therapy, it is used to prevent and treat scurvy, a disease caused by vitamin C ...
Vitamin A deficiency affects one third of children under age 5 around the world, [33] leading to 670,000 deaths and 250,000–500,000 cases of blindness. [34] Vitamin A supplementation has been shown to reduce all-cause mortality by 12 to 24%. [35]
The historic importance of vitamin C deficiency relates to occurrence on long sea-going voyages, when the ship food supplies had no good source of the vitamin. Deficiency results in scurvy when plasma concentrations fall below 0.2 mg/dL, whereas the normal plasma concentration range is 0.4 to 1.5 mg/dL.
Organs often affected in children with kwashiorkor include the kidneys, pancreas, heart, and nervous system. [3] Other findings that may be encountered on physical exam include a distended abdomen, hair thinning, loss of teeth, skin or hair depigmentation, and dermatitis. Children with kwashiorkor often develop irritability and anorexia ...
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osteomalacia (adults), rickets (children), fractures, enthesopathy, spinal stenosis, abnormal gait, short stature, tinnitus, hearing loss, dental complications, in rare exceptions Chiari malformation can occur. Causes: A genetic mutation of the PHEX gene results in elevated FGF23 hormone. Medication: phosphate, vitamin-D or burosumab