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The diagnostic approach to hyperphosphatemia involves elucidating why phosphate entry into the extracellular fluid exceeds the degree to which it can be excreted in order to maintain normal plasma levels. A broad overview of the causes and treatment of hyperphosphatemia is presented in this topic.
Hyperphosphatemia may develop when the estimated glomerular filtration rate (eGFR) falls below 25 to 40 mL/min/1.73 m 2 [1-3]. This topic reviews recommendations regarding target serum phosphorus concentration and treatment options for hyperphosphatemia for patients with CKD.
acute treatment. Reverse underlying problem. Phosphate-restricted diet. Volume resuscitation, followed by forced diuresis using acetazolamide +/- loop diuretic. Hemodialysis may be required in severe renal dysfunction (especially in tumor lysis syndrome). chronic treatment: phos-restricted diet plus phosphate binder. Calcium acetate (Phoslo™️)
Outline the treatment and management options available for hyperphosphatemia. Describe the differential diagnosis of hyperphosphatemia. Access free multiple choice questions on this topic.
The best way to prevent hyperphosphatemia is to be aware of your kidney health and to limit the amounts of phosphate and calcium you put in your body. If you’re at risk of hyperphosphatemia, talk to a healthcare provider.
The treatment for high phosphorus depends on what is causing phosphorus to build up in your blood. If you have chronic kidney disease (CKD), talk with your doctor about what your phosphorus levels should be and whether you need to limit phosphorus in your foods, drinks or medicines.
To manage your hyperphosphatemia, you can try the following strategies. Diagnose the problem. Knowing that you have hyperphosphatemia can be the trickiest part of managing this...