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If a temporary crown becomes de-cemented, it is important that a dentist examine the patient as overeruption of the opposing teeth may prevent accurate fitting of the final crown. [8] If a dentist cannot be seen in a timely manner, the temporary crown may be re-cemented by applying temporary cement to the temporary crown.
Pericoronitis is inflammation of the soft tissues surrounding the crown of a partially erupted tooth, [1] including the gingiva (gums) and the dental follicle. [2] The soft tissue covering a partially erupted tooth is known as an operculum, an area which can be difficult to access with normal oral hygiene methods.
Temporary crowns can either be direct, if constructed by the dentist in the clinic, or indirect if they are made off-site, usually in a dental laboratory. Generally direct temporary crowns tend to be for short-term use. Where medium-term or long-term temporisation is required, the use of indirect temporary crowns should be considered. [10]
Visible loss of enamel and dentine, not TTP, normal mobility, positive to vitality testing: Tooth fragment is bonded on or restoration. Follow-up 6–8 weeks and 1 year: Smoothen sharp edges and/ or restore tooth. Review with radiographs after 6– 8 weeks. Monitor until exfoliation Complicated crown fracture: Involves the enamel, dentin and pulp
In some species, this is a normal process that occurs during the formation or maintenance of the dentition. [1] By contrast, in humans tooth ankylosis is pathological, whereby a fusion between alveolar bone and the cementum of a tooth occurs. In humans, this is a rare phenomenon in deciduous dentition and even more uncommon in permanent teeth.
Occlusal trauma; Secondary occlusal trauma on X-ray film displays two lone-standing mandibular teeth, the lower left first premolar and canine. As the remnants of a once full complement of 16 lower teeth, these two teeth have been alone in opposing the forces associated with mastication for some time, as can be evidenced by the widened PDL surrounding the premolar.
The pain pathway is mostly transmitted via myelinated Aδ (sharp or stabbing pain) and unmyelinated C nerve fibers (slow, dull, aching, or burning pain) of the trigeminal nerve, which supplies sensation to the teeth and gums via many divisions and branches. [18] Initially, pain is felt while noxious stimuli are applied (such as cold).
A degree of dentin sensitivity is normal, but pain is not usually experienced in everyday activities like drinking a cooled drink. Therefore, although the terms dentin sensitivity and sensitive dentin are used interchangeably to refer to dental hypersensitivity, [ 3 ] the latter term is the more accurate.