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The decrease in vasocongestion in post-menopausal women may require some women to use artificial sexual lubricant to avoid pain during sexual intercourse. If there is pain however, sexual drive is typically diminished or absent. Other forms of vasocongestion during human sexual activity include sex flush and the swelling of nipples in both men ...
Alpha 1 blockers inhibit vasoconstriction normally initiated by the baroreceptor reflex upon postural change and the subsequent drop in pressure. [26] Other antihypertensive medications may also cause orthostatic hypotension, in addition to anticholinergics , dopaminergic drugs, opiates and psychoactive medications .
Vasoconstriction is the narrowing of the blood vessels resulting from contraction of the muscular wall of the vessels, in particular the large arteries and small arterioles. The process is the opposite of vasodilation , the widening of blood vessels.
Vascular resistance is the resistance that must be overcome for blood to flow through the circulatory system.The resistance offered by the systemic circulation is known as the systemic vascular resistance or may sometimes be called by another term total peripheral resistance, while the resistance caused by the pulmonary circulation is known as the pulmonary vascular resistance.
This worsening acidosis along with hypoxemia, if left uncorrected, eventually causes the loss of peripheral vasoconstriction, worsening hemodynamic compromise, and death. [3] The body's compensation varies by cardiopulmonary comorbidities, age, and vasoactive medications. Due to these factors, heart rate and blood pressure responses are ...
Experiments have shown that three other vascular responses to immersion of the finger in cold water are possible: a continuous state of vasoconstriction; slow, steady, and continuous rewarming; and a proportional control form in which the blood vessel diameter remains constant after an initial phase of vasoconstriction.
Vasodilation and vasoconstriction are complex phenomena; they are functions not merely of the fluid mechanics of pressure and tissue elasticity but also of active homeostatic regulation with hormones and cell signaling, in which the body produces endogenous vasodilators and vasoconstrictors to modify its vessels' compliance.
Many of the genes repressed during cold shock are involved in cell metabolism. By knowing the mechanism by which these genes respond, one can potentially tune it, in genetically modified bacteria, to modify at which temperature is the response to cold shock activated. This modification could reduce the energy costs of bioreactors. [11]