Ad
related to: frontal lobe shrinkage symptoms
Search results
Results From The WOW.Com Content Network
Frontal lobe syndrome can be caused by a range of conditions including head trauma, tumours, neurodegenerative diseases, neurodevelopmental disorders, neurosurgery and cerebrovascular disease. Frontal lobe impairment can be detected by recognition of typical signs and symptoms, use of simple screening tests, and specialist neurological testing.
An overlap between symptoms can occur as the disease progresses and spreads through the brain regions. [14] Structural MRI scans often reveal frontal lobe and/or anterior temporal lobe atrophy, but in early cases the scan may seem normal. Atrophy can be either bilateral or asymmetric. [13]
Increased alcohol intake is associated with damage to brain regions including the frontal lobe, [3] limbic system, and cerebellum, [4] with widespread cerebral atrophy, or brain shrinkage caused by neuron degeneration. This damage can be seen on neuroimaging scans. [5]
Neuroimaging studies also show that cortical shrinkage in "uncomplicated alcoholism" is most severe in the frontal lobe in comparison to the other divisions of the cerebral cortex. [15] In addition, neurological diseases that co-occur with excessive alcohol consumption—such as Wernicke–Korsakoff syndrome (WKS)—are characterized by ...
Neuroimaging evidence of cerebellar atrophy, especially in the vermis. C. The following clinical features cast doubt on the diagnosis of alcohol-related dementia: The presence of language impairment, especially dysnomia or anomia. the presence of focal neurologic signs or symptoms (except ataxia or peripheral sensory polyneuropathy).
The most frequent cause of the syndrome is brain damage to the frontal lobe. Brain damage leading to the dysexecutive pattern of symptoms can result from physical trauma such as a blow to the head or a stroke [6] or other internal trauma. It is important to note that frontal lobe damage is not the only cause of the syndrome.
It is characterized by atrophy in the frontal lobe and temporal lobe of the brain, with sparing of the parietal and occipital lobes. [1] [2] Common proteinopathies that are found in FTLD include the accumulation of tau proteins and TAR DNA-binding protein 43 (TDP-43).
the frontal lobe. In addition to the damage seen in these areas there have been reports of damage to cortex , although it was noted that this may be due to the direct toxic effects of alcohol as opposed to thiamine deficiency that has been attributed as the underlying cause of Wernicke-Korsakoff Syndrome.