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The reason behind Buster’s drastic change is vitiligo, a genetic condition often seen in humans but rarely documented in canines. This condition leads to the loss of […]
Incidences of autoimmune skin diseases can vary based on several factors. Some of these factors include the breed, age, and sex of the dog, along with individual genetics and environmental factors. [3] In terms of vitiligo, certain breeds of dogs have a higher chance of developing the disease based on their genetics.
My dog Buster has the skin condition vitiligo. It causes depigmentation of the skin (and fur). These photos beautifully capture how vitiligo has made Buster even more unique: November 2021 ...
Research indicates that the majority of variation in coat growth pattern, length and curl can be attributed to mutations in four genes, the R-spondin-2 gene or RSPO2, the fibroblast growth factor-5 gene or FGF5, the keratin-71 gene or KRT71 [15] and the melanocortin 5 receptor gene (MC5R). The wild-type coat in dogs is short, double and straight.
Some genetic conditions that result in a "leucistic" appearance include piebaldism, Waardenburg syndrome, vitiligo, Chédiak–Higashi syndrome, flavism, isabellinism, xanthochromism, axanthism, amelanism, and melanophilin mutations. Pale patches of skin, feathers, or fur (often referred to as "depigmentation") can also result from injury.
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A recessive mutation in this E gene truncates the protein, producing a non-functional receptor incapable of directing eumelanin deposition in the fur. [16] Among dogs, this mutation is unique to yellow Labrador Retrievers and Golden Retrievers and is thought to have arisen in the retriever population before these individual breeds became distinct.
The condition is very common in mice, rabbits, dogs, sheep, deer, cattle and horses—where selective breeding has increased the incidence of the mutation—but occurs among chimpanzees and other primates only as rarely as among humans. Piebaldism is unrelated to conditions such as vitiligo or poliosis.