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  2. Hepatic encephalopathy - Wikipedia

    en.wikipedia.org/wiki/Hepatic_encephalopathy

    Hepatic encephalopathy is possibly reversible with treatment. [1] This typically involves supportive care and addressing the triggers of the event. [4] Lactulose is frequently used to decrease ammonia levels. [1] Certain antibiotics (such as rifaximin) and probiotics are other potential options. [1]

  3. Acute liver failure - Wikipedia

    en.wikipedia.org/wiki/Acute_liver_failure

    One scheme defines "acute hepatic failure" as the development of encephalopathy within 26 weeks of the onset of any hepatic symptoms. This is sub-divided into "fulminant hepatic failure", which requires onset of encephalopathy within 8 weeks, and "subfulminant", which describes onset of encephalopathy after 8 weeks but before 26 weeks. [24]

  4. Liver failure - Wikipedia

    en.wikipedia.org/wiki/Liver_failure

    One scheme defines "acute hepatic failure" as the development of encephalopathy within 26 weeks of the onset of any hepatic symptoms. This is sub-divided into "fulminant hepatic failure", which requires onset of encephalopathy within 8 weeks, and "subfulminant", which describes onset of encephalopathy after 8 weeks but before 26 weeks. [6]

  5. Cirrhosis - Wikipedia

    en.wikipedia.org/wiki/Cirrhosis

    Hepatic encephalopathy is a potential complication of cirrhosis. [33] It may lead to functional neurological impairment ranging from mild confusion to coma. [33] Hepatic encephalopathy is primarily caused by the accumulation of ammonia in the blood, which causes neurotoxicity when crossing the blood-brain barrier.

  6. Fetor hepaticus - Wikipedia

    en.wikipedia.org/wiki/Fetor_hepaticus

    Fetor hepaticus or foetor hepaticus (Latin, "liver stench" ("fetid liver") [1] (see spelling differences), also known as breath of the dead or hepatic foetor, is a condition seen in portal hypertension where portosystemic shunting allows thiols to pass directly into the lungs.

  7. Encephalopathy - Wikipedia

    en.wikipedia.org/wiki/Encephalopathy

    Ifosfamide can cause a severe encephalopathy (but it can be reversible with stopping use of the drug and starting the use of methylene blue). [5] Bevacizumab and other anti–vascular endothelial growth factor medication can cause posterior reversible encephalopathy syndrome. [5]

  8. Alcoholic liver disease - Wikipedia

    en.wikipedia.org/wiki/Alcoholic_liver_disease

    Continuation of alcohol use will result in a higher risk of progression of liver disease and cirrhosis. In patients with acute alcoholic hepatitis, clinical manifestations include fever, jaundice, hepatomegaly, and possible hepatic decompensation with hepatic encephalopathy, variceal bleeding, and ascites accumulation. Tender hepatomegaly may ...

  9. Liver support system - Wikipedia

    en.wikipedia.org/wiki/Liver_support_system

    Classification for hepatic insufficiency. In hyperacute and acute liver failure, the clinical picture develops rapidly with progressive encephalopathy and multiorgan dysfunction such as hyperdynamic circulation, coagulopathy, acute kidney injury and respiratory insufficiency, severe metabolic alterations, and cerebral edema that can lead to brain death.

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