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  2. Lipoprotein lipase deficiency - Wikipedia

    en.wikipedia.org/wiki/Lipoprotein_lipase_deficiency

    The diagnosis of familial lipoprotein lipase deficiency is finally confirmed by detection of either homozygous or compound heterozygous pathogenic gene variants in LPL with either low or absent lipoprotein lipase enzyme activity. [citation needed] Lipid measurements · Milky, lipemic plasma revealing severe hyperchylomicronemia; [citation needed]

  3. Hypolipoproteinemia - Wikipedia

    en.wikipedia.org/wiki/Hypolipoproteinemia

    It can be diagnosed via blood study that identifies fat particles. The patient must fast overnight to prevent interference from fat in the blood due to food intake. The criteria for this (without the involvement of cholesterol-lowering drugs) are total cholesterol levels below 120 mg/dL and LDL cholesterol levels under 50 mg/dL. [1]

  4. Hyperlipidemia - Wikipedia

    en.wikipedia.org/wiki/Hyperlipidemia

    Lipoprotein lipase deficiency (type Ia), due to a deficiency of lipoprotein lipase (LPL) or altered apolipoprotein C2, resulting in elevated chylomicrons, the particles that transfer fatty acids from the digestive tract to the liver; Familial apoprotein CII deficiency (type Ib), [17] [18] a condition caused by a lack of lipoprotein lipase ...

  5. Familial hypertriglyceridemia - Wikipedia

    en.wikipedia.org/wiki/Familial_hypertriglyceridemia

    Inactivity of lipoprotein lipase (LPL) plays the predominant role in the development of familial hypertriglyceridemia. LPL plays a role in the metabolism of triglycerides within VLDL molecules. Inactivation mutations in LPL will create an environment with an increased concentration of VLDL molecules and therefore, triglycerides.

  6. Lipoprotein lipase - Wikipedia

    en.wikipedia.org/wiki/Lipoprotein_lipase

    Lipoprotein lipase has been shown to interact with LRP1. [ 51 ] [ 52 ] [ 53 ] It is also a ligand for α2M , GP330 , and VLDL receptors. [ 23 ] LPL has been shown to be a ligand for LRP2 , albeit at a lower affinity than for other receptors; however, most of the LPL-dependent VLDL degradation can be attributed to the LRP2 pathway. [ 23 ]

  7. Familial dysbetalipoproteinemia - Wikipedia

    en.wikipedia.org/.../Familial_dysbetalipoproteinemia

    ApoE serves as a ligand for the liver receptor for chylomicrons, IDL and VLDL, also known as very-low-density-lipoprotein receptor. Individuals with two copies of the ApoE2 gene have substantially elevated risk of this condition.

  8. Hypoalphalipoproteinemia - Wikipedia

    en.wikipedia.org/wiki/Hypoalphalipoproteinemia

    Download as PDF; Printable version ... Hypoalphalipoproteinemia is a high-density lipoprotein deficiency, ... Lecithin cholesterol acyltransferase deficiency (LCAT ...

  9. Lipaemia retinalis - Wikipedia

    en.wikipedia.org/wiki/Lipaemia_retinalis

    Lipaemia retinalis (LR) also spelled as Lipemia retinalis is an eye disease caused by high amounts of triglycerides in the blood (hypertriglyceridemia) or Lipoprotein lipase deficiency (chylomicronemia). In this condition the retinal arteries and veins, and occasionally the entire fundus shows creamy-white to salmon red discoloration.