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Cervical spinal stenosis is one of the most common forms of spinal stenosis, along with lumbar spinal stenosis (which occurs at the level of the lower back instead of the neck). Thoracic spinal stenosis, at the level of the mid-back, is much less common. [2] Cervical spinal stenosis can be far more dangerous by compressing the spinal cord.
They allow for flexion and extension and limit lateral flexion in the cervical spine. Pathological processes that can occur in these joints include degenerative changes or hypertrophic arthritis, resulting in foraminal stenosis and nerve compression. Foraminal stenosis at this joint is the most common cause of cervical nerve root pressure.
Such severe spinal stenosis symptoms are virtually absent in lumbar stenosis, however, as the spinal cord terminates at the top end of the adult lumbar spine, with only nerve roots (cauda equina) continuing further down. [15] Cervical spinal stenosis is a condition involving narrowing of the spinal canal at the level of the neck.
Within the cervical spine, most joints are innervated by the medial branch nerve (a branch of the dorsal rami) from the same levels. In other words, the facet joint between C4 and C5 vertebral segments is innervated by the C4 and C5 medial branch nerves. However, there are two exceptions:
Occipito-cervical junction This disorder may result from rheumatoid arthritis, causing the hypermobility of the connection between the neck and head, resulting in paralysis or pain. [6] Cerebrovascular disease Cerebrovascular disease is a type of cervical spine disorder that can cause tetraplegia. [7] Subaxial cervical spine [8] Atlanto-axial joint
Cervical spinal nerve 4, also called C4, is a spinal nerve of the cervical segment. It originates from the spinal cord above the 4th cervical vertebra (C4). It contributes nerve fibers to the phrenic nerve , the motor nerve to the thoracoabdominal diaphragm .
These changes are more pronounced as time progresses after injury, and are evidenced by end plate osteophytosis, disc damage, disc narrowing, desiccation and disc bulging. “A retrolisthesis hyperloads at least one disc and puts shearing forces on the anterior longitudinal ligament, the annular rings, nucleus pulposus , cartilage end plates ...
These conditions contribute to the potential narrowing of the spinal cord, increasing pressure and inducing damage on the spinal nerve roots, thus, causing paing, tingling or weakness in the lower body. [5] Risk factors for LSS include: [16] [15] Age; Degenerative changes of the spine; Obesity; Family history of spinal stenosis; Tobacco use