When.com Web Search

Search results

  1. Results From The WOW.Com Content Network
  2. NMDA receptor modulator - Wikipedia

    en.wikipedia.org/wiki/NMDA_receptor_modulator

    The first compound studied was glycine which was hypothesized by Daniel Javitt after observation that people with phencyclidine(PCP)-induced psychosis were lacking in glutamate transmission. [1] (PCP is an NMDA receptor antagonist that blocks glutamate.) In giving glycine to people with PCP-induced psychosis a recovery rate was noted.

  3. Glycine - Wikipedia

    en.wikipedia.org/wiki/Glycine

    Glycine (symbol Gly or G; [6] / ˈ ɡ l aɪ s iː n / ⓘ) [7] is an amino acid that has a single hydrogen atom as its side chain. It is the simplest stable amino acid (carbamic acid is unstable). Glycine is one of the proteinogenic amino acids. It is encoded by all the codons starting with GG (GGU, GGC, GGA, GGG). [8]

  4. Glycine receptor - Wikipedia

    en.wikipedia.org/wiki/Glycine_receptor

    The receptor can be activated by a range of simple amino acids including glycine, β-alanine and taurine, and can be selectively blocked by the high-affinity competitive antagonist strychnine. [2] Caffeine is a competitive antagonist of GlyR. [3] Cannabinoids enhance the function. [4]

  5. Sodium- and chloride-dependent glycine transporter 2

    en.wikipedia.org/wiki/Sodium-_and_chloride...

    Sodium- and chloride-dependent glycine transporter 2, also known as glycine transporter 2 (GlyT2), is a protein that in humans is encoded by the SLC6A5 gene. [ 5 ] The glycine transporter 2 is a membrane protein which recaptures glycine , a major inhibitory transmitter in the spinal cord and brainstem .

  6. Glycine transporter - Wikipedia

    en.wikipedia.org/wiki/Glycine_transporter

    Glycine transporters (GlyTs) are plasmalemmal neurotransmitter transporters. They serve to terminate the signaling of glycine by mediating its reuptake from the synaptic cleft back into the presynaptic neurons .

  7. Hyperglycinemia - Wikipedia

    en.wikipedia.org/wiki/Hyperglycinemia

    Hyperglycinemia may refer to one of two related inborn amino acid disorders that are characterized by elevated levels of glycine in the blood. Propionic acidemia, also known as "ketotic glycinemia" Glycine encephalopathy, also known as "non-ketotic hyperglycinemia"

  8. Hyperekplexia - Wikipedia

    en.wikipedia.org/wiki/Hyperekplexia

    The three main signs of hyperekplexia are generalized stiffness, excessive startle response beginning at birth, and nocturnal myoclonus. [5] Affected individuals are fully conscious during episodes of stiffness, which consist of forced closure of the eyes and an extension of the extremities followed by a period of generalised stiffness and uncontrolled falling at times. [6]

  9. Glycinergic - Wikipedia

    en.wikipedia.org/wiki/Glycinergic

    A glycinergic agent (or drug) is a chemical which functions to directly modulate the glycine system in the body or brain. Examples include glycine receptor agonists, glycine receptor antagonists, and glycine reuptake inhibitors.