Search results
Results From The WOW.Com Content Network
Damage to the auditory cortex in humans leads to a loss of any awareness of sound, but an ability to react reflexively to sounds remains as there is a great deal of subcortical processing in the auditory brainstem and midbrain. [13] [14] [15] Neurons in the auditory cortex are organized according to the frequency of sound to which they respond ...
Cortical deafness is a rare form of sensorineural hearing loss caused by damage to the primary auditory cortex.Cortical deafness is an auditory disorder where the patient is unable to hear sounds but has no apparent damage to the structures of the ear (see auditory system).
The auditosensory cortex is the part of the auditory system that is associated with the sense of hearing in humans. It occupies the bilateral primary auditory cortex in the temporal lobe of the mammalian brain. [1]
They suggested that pre-phonemic auditory agnosia manifests because of general damage to the auditory cortex of both hemispheres, and that post-phonemic auditory agnosia manifests because of damage to a spoken word recognition center in the left hemisphere. A recent research on an epileptic patient supported this hypothesis.
Finally, the signal goes to the auditory cortex of the temporal lobe to be interpreted as sound. [83] Hearing loss is most commonly caused by long-term exposure to loud noises, from recreation or from work, that damage the hair cells, which do not grow back on their own. [84] [85] [9]
Wernicke's area receives information from the auditory cortex, and functions to assign word meanings. [19] This is why damage to this area results in meaningless speech, often with paraphasic errors and newly created words or expressions.
The results showed far less activity in the auditory nerve, as well as hyperactivity in the brainstem among participants with tinnitus compared to participants with “normal hearing,” Maison said.
Structural damage to hair cells (primarily the outer hair cells) will result in hearing loss that can be characterized by an attenuation and distortion of incoming auditory stimuli. During hair cell death 'scars' develop, which prevent potassium rich fluid of the endolymph from mixing with the fluid on the basal domain. [ 86 ]