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Selenium is toxic in high concentrations. As sodium selenite, the chronic toxic dose for human beings was described as about 2.4 to 3 milligrams of selenium per day. [7] In 2000, the US Institute of Medicine set the adult Tolerable upper intake levels (UL) for selenium from all sources - food, drinking water and dietary supplements - at 400 μg/day. [8]
Sodium hydrogen selenite. Sodium hydrogen selenite is an inorganic chemical consisting of a ratio of one hydrogen, one sodium, three oxygen, and one selenium atom. It is the sodium salt of the conjugate base of selenous acid. This compound finds therapeutic application for providing the essential trace element selenium.
Sodium selenate is produced by oxidation of selenium, first with nitric acid, producing selenous acid. The selenous acid is neutralized to form sodium selenite. The sodium selenite is oxidized in a basic medium hydrogen peroxide to form a selenate, which is then spray-dried. [3] Se + 2HNO 3 → H 2 SeO 3 + NO + NO 2 H 2 SeO 3 + Na 2 CO 3 → Na ...
These include silver selenite (Ag 2 SeO 3) and sodium selenite (Na 2 SeO 3). Hydrogen sulfide reacts with aqueous selenous acid to produce selenium disulfide : H 2 SeO 3 + 2 H 2 S SeS 2 + 3 H 2 O {\displaystyle {\ce {H2SeO3 + 2 H2S -> SeS2 + 3 H2O}}}
Spinal cord injury research seeks new ways to cure or treat spinal cord injury in order to lessen the debilitating effects of the injury in the short or long term. There is no cure for SCI, and current treatments are mostly focused on spinal cord injury rehabilitation and management of the secondary effects of the condition. [1]
In Pakistan, spinal cord injury is more common in males (92.68%) as compared to females in the 20–30 years of age group with a median age of 40 years, although people from 12–70 years of age suffered from spinal cord injury [73] Rates of injury are at their lowest in children, at their highest in the late teens to early twenties, then get ...
Move around as soon as possible after having been confined to bed, such as after surgery, illness, or injury. If you're at risk for DVT, talk to your doctor about: Graduated compression stockings (sometimes called "medical compression stockings") Medication (anticoagulants) to prevent DVT.
The NaV1.7 and NaV1.8 sodium channels are found in nerve cells called nociceptors, that transmit pain signals to the spinal cord and brain. The SCN9A gene mutations which cause small fiber neuropathy result in NaV1.7 sodium channels that do not close completely when the channel is turned off.