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  2. Acephate - Wikipedia

    en.wikipedia.org/wiki/Acephate

    Acephate is an organophosphate foliar and soil insecticide of moderate persistence with residual systemic activity of about 10–15 days at the recommended use rate. It is used primarily for control of aphids, including resistant species, in vegetables (e.g. potatoes, carrots, greenhouse tomatoes, and lettuce) and in horticulture (e.g. on roses and greenhouse ornamentals).

  3. Imidacloprid - Wikipedia

    en.wikipedia.org/wiki/Imidacloprid

    Imidacloprid is a systemic insecticide, belonging to the class of chloronicotinyl neonicotinoid insecticides. It works by interfering with the transmission of nerve impulses in insects by binding irreversibly to specific insect nicotinic acetylcholine receptors. [28] It is in IRAC group 4A.

  4. Neonicotinoid - Wikipedia

    en.wikipedia.org/wiki/Neonicotinoid

    It is systemic with particular efficacy against sucking insects and has a long residual activity. Imidacloprid can be added to the water used to irrigate plants. Controlled-release formulations of imidacloprid take 2–10 days to release 50% of imidacloprid in water. [36]

  5. Terbufos - Wikipedia

    en.wikipedia.org/wiki/Terbufos

    Terbufos is a chemical compound used in insecticides and nematicides. It is part of the chemical family of organophosphates. It is a clear, colourless to pale yellow or reddish-brown liquid and sold commercially as granulate. [5]

  6. Nitenpyram - Wikipedia

    en.wikipedia.org/wiki/Nitenpyram

    Nitenpyram is highly selective towards the variation of the nACHr which insects possess, and has seen extensive use in targeted, insecticide applications. Known under the codename TI 304 during field testing starting in 1989, the compound's first documented commercial use was in 1995 under the name "Bestguard" as an agricultural insecticide. [1]

  7. Fipronil - Wikipedia

    en.wikipedia.org/wiki/Fipronil

    Fipronil is a broad-spectrum insecticide that belongs to the phenylpyrazole insecticide class. [3] Fipronil disrupts the insect central nervous system by blocking the ligand-gated ion channel of the GABA A receptor (IRAC group 2B) and glutamate-gated chloride (GluCl) channels. This causes hyperexcitation of contaminated insects' nerves and muscles.