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  2. Glucogenic amino acid - Wikipedia

    en.wikipedia.org/wiki/Glucogenic_amino_acid

    Alanine is a glucogenic amino acid that the liver's gluconeogenesis process can use to produce glucose. Muscle cells break down their protein when their blood glucose levels fall, which happens during fasting or periods of intense exercise. The breakdown process releases alanine, which is then transferred to the

  3. Protein metabolism - Wikipedia

    en.wikipedia.org/wiki/Protein_metabolism

    Protein anabolism is the process by which proteins are formed from amino acids. It relies on five processes: amino acid synthesis, transcription, translation, post translational modifications, and protein folding. Proteins are made from amino acids. In humans, some amino acids can be synthesized using already existing intermediates. These amino ...

  4. Anabolism - Wikipedia

    en.wikipedia.org/wiki/Anabolism

    Photosynthetic carbohydrate synthesis in plants and certain bacteria is an anabolic process that produces glucose, cellulose, starch, lipids, and proteins from CO 2. [6] It uses the energy produced from the light-driven reactions of photosynthesis, and creates the precursors to these large molecules via carbon assimilation in the photosynthetic ...

  5. Metabolic window - Wikipedia

    en.wikipedia.org/wiki/Metabolic_window

    The net muscle protein balance (NBAL) is the relationship between muscle protein breakdown and muscle protein synthesis. It is determined by the stability between the two processes. [1] The anabolic state theory suggests that it is critical to consume proteins and carbohydrates immediately after resistance training to increase muscle protein ...

  6. Glycogen storage disease - Wikipedia

    en.wikipedia.org/wiki/Glycogen_storage_disease

    A glycogen storage disease (GSD, also glycogenosis and dextrinosis) is a metabolic disorder caused by a deficiency of an enzyme or transport protein affecting glycogen synthesis, glycogen breakdown, or glucose breakdown, typically in muscles and/or liver cells. [1] GSD has two classes of cause: genetic and environmental.

  7. Catabolism - Wikipedia

    en.wikipedia.org/wiki/Catabolism

    Released from alpha cells in the pancreas either when starving or when the body needs to generate additional energy; it stimulates the breakdown of glycogen in the liver to increase blood glucose levels; its effect is the opposite of insulin; glucagon and insulin are a part of a negative-feedback system that stabilizes blood glucose levels ...

  8. Purine nucleotide cycle - Wikipedia

    en.wikipedia.org/wiki/Purine_nucleotide_cycle

    Myogenic hyperuricemia, as a result of the purine nucleotide cycle running when ATP reservoirs in muscle cells are low (ADP > ATP), is a common pathophysiologic feature of glycogenoses such as GSD-III, GSD-V and GSD-VII, as they are metabolic myopathies which impair the ability of ATP (energy) production within muscle cells.

  9. Myokine - Wikipedia

    en.wikipedia.org/wiki/Myokine

    The definition and use of the term myokine first occurred in 2003. [5] In 2008, the first myokine, myostatin, was identified. [4] [6] The gp130 receptor cytokine IL-6 (Interleukin 6) was the first myokine found to be secreted into the blood stream in response to muscle contractions. [7] [8]