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  2. Aleniglipron - Wikipedia

    en.wikipedia.org/wiki/Aleniglipron

    Aleniglipron (development code GSBR-1290) is a small-molecule GLP-1 agonist developed by Structure Therapeutics. [1] It is delivered orally and is in a Phase II trial as of 2023.

  3. GLP-1 receptor agonist - Wikipedia

    en.wikipedia.org/wiki/GLP-1_receptor_agonist

    Attempts to develop an orally bioavailable GLP-1 agonist, either a modified peptide, as in the case of oral semaglutide, [34] or a small molecule drug, have produced additional drug candidates. [26] Other companies have tested inhaled or transdermal administration.

  4. Orforglipron - Wikipedia

    en.wikipedia.org/wiki/Orforglipron

    Orforglipron (LY-3502970) is an oral, non-peptide, small molecule glucagon-like peptide-1 receptor agonist developed as a weight loss drug by Eli Lilly and Company. [1] [2] It is easier to produce than current peptide GLP-1 agonists and is expected to be cheaper. [3]

  5. What Are GLP-1 Medications & Who Should Use Them? Here ... - AOL

    www.aol.com/glp-1-medications-them-heres...

    GLP-1 agonists are a class of medications that mimic the action of the hormone GLP-1 (glucagon-like peptide-1), which is involved in insulin production and appetite regulation. “GLP-1 stands for ...

  6. Structure Therapeutics Announces First Patients Dosed in ...

    lite.aol.com/tech/story/0022/20241113/9273366.htm

    GSBR-1290 is an orally-available, nonpeptide small molecule glucagon-like-peptide-1 receptor (GLP-1R) agonist that has demonstrated competitive weight loss and generally favorable safety and tolerability results in previous studies with once-daily dosing.

  7. Glucagon-like peptide-1 - Wikipedia

    en.wikipedia.org/wiki/Glucagon-like_peptide-1

    In accordance with the expression of GLP-1 receptor on brainstem and hypothalamus, GLP-1 has been shown to promote satiety and thereby reduce food and water intake. Consequently, diabetic subjects treated with GLP-1 receptor agonists often experience weight loss as opposed to the weight gain commonly induced with other treatment agents. [2] [15]

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