Search results
Results From The WOW.Com Content Network
Bile salts interfere with the gastric mucosal barrier, allowing acid to irritate the stomach lining and cause gastritis. [1] Dogs with this condition usually vomit in the morning after not eating all night. Treatment is to feed late at night. H2 blockers and antiemetics can also be used. Bilious vomiting syndrome is a diagnosis of exclusion ...
Hemorrhagic gastroenteritis (HGE) is a disease of dogs characterized by sudden vomiting and bloody diarrhea. The symptoms are usually severe, and HGE can be fatal if not treated. HGE is most common in young adult dogs of any breed, but especially small dogs such as the Toy Poodle and Miniature Schnauzer. [1] It is not contagious.
Gastric dilatation volvulus (GDV), also known as gastric dilation, twisted stomach, or gastric torsion, is a medical condition that affects dogs and rarely cats and guinea pigs, [1] in which the stomach becomes overstretched and rotated by excessive gas content. The condition also involves compression of the diaphragm and caudal vena cavae.
Acetaminophen (paracetamol, Tylenol) can cause liver damage in dogs. The toxic dose is 150 mg/kg. [174] Ibuprofen * can cause gastrointestinal irritation, stomach ulcers, and kidney damage in dogs. [175] Naproxen (Aleve)* has a long half-life in dogs and can cause gastrointestinal irritation, anemia, melena (digested blood in feces), and vomiting.
Gastritis is the inflammation of the lining of the stomach. [1] It may occur as a short episode or may be of a long duration. [1] There may be no symptoms but, when symptoms are present, the most common is upper abdominal pain (see dyspepsia). [1]
Portal hypertensive gastropathy can also be treated with endoscopic treatment delivered through a fibre-optic camera into the stomach. Argon plasma coagulation and electrocautery have both been used to stop bleeding from ectatic vessels, and to attempt to obliterate the vessels, but have limited utility if the disease is diffuse.
Gastric chief cells are generally located deep in the mucosal layer of the stomach lining, in the fundus and body of the stomach. [2] [3] Chief cells release the zymogen (enzyme precursor) pepsinogen when stimulated by a variety of factors including cholinergic activity from the vagus nerve and acidic condition in the stomach.
Gastric motor function abnormalities have long been linked to functional dyspepsia. [8] [9] However, a study revealed that there was no relationship between symptoms and stomach physiological abnormalities. [10] The symptoms are significantly influenced by meal consumption, [11] and genetic factors may also play a part. [12]