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Bromethalin works by being metabolised to n-desmethyl-bromethalin and uncoupling mitochondrial oxidative phosphorylation, which causes a decrease in adenosine triphosphate (ATP) synthesis. The decreased ATP inhibits the activity of the Na/K ATPase enzyme , thereby leading to a subsequent buildup of cerebral spinal fluid (CSF) and vacuolization ...
For the fast-acting rodenticide bromethalin, for example, there is no diagnostic test or antidote. [ 23 ] This has led environmental researchers to conclude that low strength, long duration rodenticides (generally first generation anticoagulants) are the best balance between maximum effect and minimum risk.
Warning label on a tube of rat poison containing bromadiolone on a dike of the Scheldt river in Steendorp, Belgium. Bromadiolone is a potent anticoagulant rodenticide.It is a second-generation 4-hydroxycoumarin derivative and vitamin K antagonist, often called a "super-warfarin" for its added potency and tendency to accumulate in the liver of the poisoned organism.
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For this purpose, baits containing anticoagulants such as Warfarin were long used; they kill relatively slowly through internal bleeding, which is not associated with ingestion. More recently a highly potent toxin attacking the central nervous system, bromethalin, has been used. Again, with sub-lethal doses of this chemical, the animal cannot ...
Secondary poisoning, or relay toxicity, is the poisoning that results when one organism comes into contact with or ingests another organism that has poison in its system. It typically occurs when a predator eats an animal, such as a mouse, rat, or insect, that has previously been poisoned by a commercial pesticide.
Phenylsilatrane is a convulsant chemical which has been used as a rodenticide. [1] [2] Phenylsilatrane and some of its analogs with 4-substituents of H, CH 3, Cl, Br, and CSi(CH 3) 3 are highly toxic to mice.
Brodifacoum is a 4-hydroxycoumarin anticoagulant, with a similar mode of action to its historical predecessors dicoumarol and warfarin.However, due to very high potency and long duration of action (elimination half-life of 20 – 130 days), it is characterised as a "second-generation" or "superwarfarin" anticoagulant.