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Keratosis pilaris (KP; also follicular keratosis, lichen pilaris, or colloquially chicken skin. [ 1 ] ) is a common, autosomal - dominant , genetic condition of the skin's hair follicles characterized by the appearance of possibly itchy , small, gooseflesh -like bumps, with varying degrees of reddening or inflammation. [ 2 ]
Sarcoidosis, an inflammatory disease, involves the skin in about 25% of patients. The most common lesions are erythema nodosum, plaques, maculopapular eruptions, subcutaneous nodules, and lupus pernio. Treatment is not required, since the lesions usually resolve spontaneously in two to four weeks.
The skin weighs an average of four kilograms, covers an area of two square metres, and is made of three distinct layers: the epidermis, dermis, and subcutaneous tissue. [1] The two main types of human skin are: glabrous skin, the hairless skin on the palms and soles (also referred to as the "palmoplantar" surfaces), and hair-bearing skin. [3]
Ringworm. What it looks like: Ringworm is a common skin infection caused by a fungus. It gets its name from its circular rash, which is often red, swollen, and cracked. Other symptoms to note ...
Color Change: The affected area turns brown, then darkens to a black or purplish color. Dryness: The skin becomes dry and shriveled. Coldness: The affected area may feel cold to the touch. Loss of Sensation: Numbness or a loss of feeling in the affected area. Odor: Typically no odor, as there is no infection involved, but the tissue itself is dead.
Schamberg's disease, (also known as "progressive pigmentary dermatosis of Schamberg", [1] "purpura pigmentosa progressiva" (PPP), [1] and "Schamberg's purpura" [1]) is a chronic discoloration of the skin found in people of all ages, usually only affecting the feet, legs or thighs or a combination.
Drug-induced pigmentation of the skin may occur as a consequence of drug administration, and the mechanism may be postinflammatory hyperpigmentation in some cases, but frequently is related to actual deposition of the offending drug in the skin. [2]: 125–6 The incidence of this change varies, and depends on the type of medication involved.
Initially, the skin turns red and starts to itch and burn. Large blisters (or bullae) form within 48 hours. [8] The blisters may leave black, brown, or purplish scars that can last for several years. This hyperpigmentation of the skin is caused by the production of melanin triggered by the furanocoumarins.