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The T helper type 2 predominant airways disease caused by M. pneumoniae infection may lead to IgE-related hyper-responsiveness and eosinophil function, resulting in an onset of asthma. [13] There is also a possibility that M. pneumoniae infection may destroy respiratory mucosal cells and facilitate the penetration of antigens into the mucosa. [13]
However, asthma is rarely the only symptom, and not all people with food or other allergies have asthma [17] Sulfite sensitivity Asthma can occur in reaction to ingestion or inhalation of sulfites, which are added to foods and wine as preservatives. [18] Salicylate sensitivity Salicylates can trigger asthma in sensitive individuals. Salicylates ...
The simple explanation for allergic inflammation in asthma begins with the development of a predominantly helper T2 lymphocyte–driven, as opposed to helper T1 lymphocyte–driven, immune milieu, perhaps caused by certain types of immune stimulation early in life. This is followed by allergen exposure in a genetically susceptible individual.
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In the study, 27% of the children had an upper respiratory infection, and the children who had double the cockroach concentration had an 18% increased risk of upper respiratory infection; a 31% ...
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