Search results
Results From The WOW.Com Content Network
Hypernatremia due to diabetes insipidus as a result of a brain disorder, may be treated with the medication desmopressin. [1] If the diabetes insipidus is due to kidney problems the medication causing the problem may need to be stopped or the underlying electrolyte disturbance corrected. [1] [7] Hypernatremia affects 0.3–1% of people in ...
[3] [27] Once the patient is stable, it is important to identify the underlying cause of hypernatremia as that may affect the treatment plan. [3] [27] The final step in treatment is to calculate the patients free water deficit, and to replace it at a steady rate using a combination of oral or IV fluids.
Studies that examine the cognitive development of children stress the importance of brain development and the presence of a stimulating environment to develop cognitive skills. [ 18 ] [ 19 ] The rapid brain development in the first years of life is primarily responsible for the infants' ability to process visual information, tactile information ...
A study has revealed that serum progesterone concentrations in boys (10 days to 18 years old) with 21-hydroxylase deficiency reached levels up to 10.14 ng/mL, i.e. similar to female luteal values, while in the control group of boys average level was 0.07 ng/mL (0.22 nmol/L), with values ranging from 0.05 to 0.40 ng/mL. [38]
Cortisol at high concentrations can cross-react and activate the mineralocorticoid receptor due to the non-selectivity of the receptor, leading to aldosterone-like effects in the kidney. This is what causes the hypokalemia, hypertension, and hypernatremia associated with the syndrome. Patients often present with severe hypertension and end ...
An ADA spokesperson, Dr. Scott Tomar, said that if more robust studies prove a link between fluoride and brain development, public health leaders should take a closer look at the mineral's effects ...
The association of brain edema with increased in hospital risk of death was observed in TBI across all level of severity. [34] Edema in the acute and chronic phases were associated with a worse neurologic and clinical outcome. [34] Children with TBI and cerebral edema have worse clinical outcomes as well. [34]
The development of memory in children becomes evident within the first 2 to 3 years of a child's life as they show considerable advances in declarative memory. This enhancement continues into adolescence with major developments in short term memory, working memory, long-term memory and autobiographical memory. [21]