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One of the first "eradication protocols", if not the first, was used by J. Robin Warren and Barry Marshall. Barry Marshall treated his own gastritis, which developed following intentional ingestion of H. pylori culture. He used bismuth salt and metronidazole. This treatment effectively cured his gastritis and eliminated the H. pylori infection ...
First line therapy is recommended if low-grade gastric MALT lymphoma is diagnosed, regardless of evidence of H. pylori. However, if a severe condition of atrophic gastritis with gastric lesions is reached antibiotic-based treatment regimens are not advised since such lesions are often not reversible and will progress to gastric cancer. [46]
Antacids are a common treatment for mild to medium gastritis. [29] When antacids do not provide enough relief, medications such as H 2 blockers and proton-pump inhibitors that help reduce the amount of acid are often prescribed. [29] [30] Cytoprotective agents are designed to help protect the tissues that line the stomach and small intestine. [31]
First report of resistance of H. pylori to the antibiotic metronidazole. [52] Resistance of H. pylori to treatment will lead to the development of many different antibiotic and proton pump inhibitor regimens for eradication. [53] 1992 Fukuda et al. prove ingestion of H. pylori causes gastritis in rhesus monkeys. [4]
Sucralfate is used for the treatment of active duodenal ulcers not related to the use of nonsteroidal anti-inflammatory drugs (NSAIDs), as the mechanism behind these ulcers is due to acid oversecretion. [1] It is not FDA approved for gastric ulcers, but is widely used because of evidence of efficacy. [10]
Stress gastritis and ulcer prevention in critical care [18] Gastrinomas; Zollinger–Ellison syndrome (often 2–3× the regular dose is required) [19] Specialty professional organizations recommend that people take the lowest effective PPI dose to achieve the desired therapeutic result when used to treat gastroesophageal reflux disease long-term.