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The regimen also improves or normalizes lung function in cases of pulmonary alveolar proteinosis and pulmonary artery hypertension and may halt the progression or improve the function of other organs directly injured by GATA2 deficiency. [19] [15] [9]
2034 13819 Ensembl ENSG00000116016 ENSMUSG00000024140 UniProt Q99814 P97481 RefSeq (mRNA) NM_001430 NM_010137 RefSeq (protein) NP_001421 NP_034267 Location (UCSC) Chr 2: 46.29 – 46.39 Mb Chr 17: 87.06 – 87.14 Mb PubMed search Wikidata View/Edit Human View/Edit Mouse Endothelial PAS domain-containing protein 1 (EPAS1, also known as hypoxia-inducible factor-2alpha (HIF-2α)) is a protein ...
[10] [11] [18] To investigate its use for heart failure in those with a preserved LVEF (HFpEF), Novartis funded the PARAGON-HF trial which was designed to investigate the use of sacubitril/valsartan in the treatment of HFpEF patients with a LVEF of 45% or more. Concluding in 2019, it failed to show significance for reducing hospitalisation ...
These patients also have vomiting and growth retardation. Kidney function is also normal if the disease is treated, [ 4 ] but occasionally patients proceed to end-stage kidney failure. Bartter syndrome consists of low levels of potassium in the blood , alkalosis, normal to low blood pressures, and elevated plasma renin and aldosterone.
Serotonin (/ ˌ s ɛr ə ˈ t oʊ n ɪ n, ˌ s ɪər ə-/) [6] [7] [8] or 5-hydroxytryptamine (5-HT) is a monoamine neurotransmitter.Its biological function is complex, touching on diverse functions including mood, cognition, reward, learning, memory, and numerous physiological processes such as vomiting and vasoconstriction.
The condition was first described as "brown lung induration" by Rudolf Virchow in 1864 in patients after their death. Wilhelm Ceelen later correlated his findings to the clinical symptoms of two children who died of IPH in 1931. [18] The first living patient was diagnosed by Jan Waldenström in 1944. [19] It has been given several names, including:
The most common cause of myocardial rupture is a recent myocardial infarction, with the rupture typically occurring three to five days after infarction. [3] Other causes of rupture include cardiac trauma, endocarditis (infection of the heart), [4] [5] cardiac tumors, infiltrative diseases of the heart, [4] and aortic dissection.