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The dorsal venous network of the hand is a venous network on the dorsum (backside) of hand. It is formed by the dorsal metacarpal veins (three in number), a dorsal digital vein from the radial (lateral) side of the index finger and one from the ulnar (medial) side of the little finger, and both dorsal digital veins of the thumb.
The pulmonary arteries carry deoxygenated blood to the lungs, where carbon dioxide is released and oxygen is picked up during respiration. [3] Arteries are further divided into very fine capillaries which are extremely thin-walled. [4] The pulmonary veins return oxygenated blood to the left atrium of the heart. [3]
On the dorsum of the foot the dorsal digital veins receive, in the clefts between the toes, the intercapitular veins from the plantar venous arch and join to form short common digital veins which unite across the distal ends of the metatarsal bones in a dorsal venous arch.
Oxygen-poor blood enters the right side of the heart through two large veins. Oxygen-rich blood from the lungs enters through the pulmonary veins on the left side of the heart into the aorta and then reaches the rest of the body. The capillaries are responsible for allowing the blood to receive oxygen through tiny air sacs in the lungs.
These are common presenting symptoms of chronic and cardiogenic pulmonary edema due to left ventricular failure. The development of pulmonary edema may be associated with symptoms and signs of "fluid overload" in the lungs; this is a non-specific term to describe the manifestations of right ventricular
Lesions lips, patient with hemorrhagic hereditary telangiectasia. Hereditary hemorrhagic telangiectasia (HHT), also known as Osler–Weber–Rendu disease and Osler–Weber–Rendu syndrome, is a rare autosomal dominant genetic disorder that leads to abnormal blood vessel formation in the skin, mucous membranes, and often in organs such as the lungs, liver, and brain.
Chronic venous insufficiency (CVI) is a medical condition characterized by blood pooling in the veins, leading to increased pressure and strain on the vein walls. [1] The most common cause of CVI is superficial venous reflux, which often results in the formation of varicose veins, a treatable condition. [2]
It is posterior to the brachial plexus, [3] and the axillary artery and vein. [4] This takes it deep to the clavicle. [2] It rests on the outer surface of the serratus anterior muscle. It extends along the side of the thorax to the lower border of the serratus anterior muscle, supplying fibres to each of the muscle's digitations. [5] [6]