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In computing, a Trojan horse (or simply Trojan) is a malware that misleads users of its true intent by disguising itself as a normal program. The term is derived from the ancient Greek story of the deceptive Trojan Horse that led to the fall of the city of Troy. [1] Trojans are generally spread by some form of social engineering.
Other viruses utilize host cell proteins to shield viral DNA until it has reached the nucleus. Upon entry into the host cell cytoplasm, the HIV-1 capsid is recognized and bound by cyclophilin A (CypA); this affinity interaction stabilizes the capsid and prevents exposure of the HIV-1 cDNA to pattern recognition receptors in the cytoplasm.
NETs allow neutrophils to kill extracellular pathogens while minimizing damage to the host cells. [3] Upon in vitro activation with the pharmacological agent phorbol myristate acetate (PMA), Interleukin 8 (IL-8) or lipopolysaccharide (LPS), neutrophils release granule proteins and chromatin to form an extracellular fibril matrix known as NET ...
Condition-based Trojans are detectable with power traces to some degree when inactive. That is due to the leakage currents generated by the trigger or counter circuit activating the Trojan. Hardware Trojans can be triggered in different ways. A Trojan can be internally activated, which means it monitors one or more signals inside the IC. The ...
Entry, or penetration, is the second step in viral replication. This step is characterized by the virus passing through the plasma membrane of the host cell. The most common way a virus gains entry to the host cell is by receptor-mediated endocytosis, which comes at no energy cost to the virus, only the host cell. Receptor-mediated endocytosis ...
To enter the cells, proteins on the surface of the virus interact with proteins of the cell. Attachment, or adsorption, occurs between the viral particle and the host cell membrane. A hole forms in the cell membrane, then the virus particle or its genetic contents are released into the host cell, where replication of the viral genome may commence.
Advantages include automatic host cell division results in replication of the virus's genes, and the fact that it is nearly impossible to remove an integrated provirus from an infected cell without killing the cell. [14] A disadvantage of this method is the need to enter the nucleus (and the need for packaging proteins that will allow for that).
How a virus enters a cell is different depending on the type of virus it is. A virus with a nonenveloped capsid enters the cell by attaching to the attachment factor located on a host cell. It then enters the cell by endocytosis or by making a hole in the membrane of the host cell and inserting its viral genome. [2]