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Type 1 second-degree heart block is considered more benign than type 2 second-degree heart block. [4] The type 1 does not have structural changes found on histology. Both types are named after Woldemar Mobitz. [5] [6] Type I is also named after Karel Frederik Wenckebach, [7] and type II is also named after John Hay. [8] [9]
By contrast, an AV block occurs in the AV node and delays ventricular depolarization. The term "Wenckebach block" is also used for some heart blocks, and can refer to a second degree type I block in either the SA node or the AV node, however the ECG features of the two are quite distinctly different.
Sinus rhythm with acute inferior infarction complicated by Type I A-V block manifest in the form of 5:4 Wenckebach periods; R-P/P-R reciprocity. Wenckebach is credited for describing the median bundle of the heart's conductive system that connects the sinoatrial node to the atrioventricular node.
The heart rate produced by the ventricles is much slower than that produced by the SA node. [1] Some AV blocks are benign, or normal, in certain people, such as in athletes or children. Other blocks are pathologic, or abnormal, and have several causes, including ischemia, infarction, fibrosis, and drugs.
A sinoatrial block (also spelled sinuatrial block) [1] is a disorder in the normal rhythm of the heart, known as a heart block, that is initiated in the sinoatrial node.The initial action impulse in a heart is usually formed in the sinoatrial node (SA node) and carried through the atria, down the internodal atrial pathways to the atrioventricular node (AV) node. [2]
These are also known as AV blocks, because the vast majority of them arise from pathology at the atrioventricular node. They are the most common causes of bradycardia: [citation needed] First-degree heart block, which manifests as PR prolongation; Second-degree heart block. Type 1 Second degree heart block, also known as Mobitz I or Wenckebach
He also contributed to Karel Frederik Wenckebach's work on what is now known as second-degree atrioventricular block of the heart in which Wenckebach described the periodicity of this block as "Luciani periodicity." [2]
This form is identical to the type of AV block described by Hay in 1906 without the benefit of electrocardiography. Mobitz included 2:1 and 3:1 AV blocks in his type II classification, and indicated the serious nature of type II block and its propensity to Adams-Stokes attacks. Sinus rhythm (rate = 100/min) with 3:2 and 2:1 Type II A-V block; RBBB