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Plummer–Vinson syndrome (also known as Paterson–Kelly syndrome [1] or Paterson–Brown-Kelly syndrome in the UK [2]) is a rare disease characterized by dysphagia (difficulty swallowing), iron-deficiency anemia, glossitis (inflammation of the tongue), cheilosis (cracking at the corners of the mouth), and esophageal webs (thin membranes in the esophagus that can cause obstruction). [1]
Deficiency of iron, folic acid or various B vitamins (glossitis e.g. due to anemia), or zinc [18] Neuropathy, e.g. following damage to the chorda tympani nerve. Hypothyroidism. Medications ("scalded mouth syndrome", unrelated to BMS) – protease inhibitors and angiotensin-converting-enzyme inhibitors (e.g. captopril). [1] [5] [13] Type 2 ...
Desiderosmia is a craving for particular sharp or pungent smells, including the smells of such inedible substances as rubber tires, menthol, gasoline, bleach and domestic cleaning products, without the desire to taste or eat the substances in question. [1] [2] Desiderosmia can be a symptom of iron deficiency. [1]
Within three weeks, she began experiencing a metallic taste and a burning sensation in her mouth that ceased when she stopped taking the medication. [48] When she began taking eprosartan on a second occasion, her dysgeusia returned. [48] In a second case, a fifty-nine-year-old man was prescribed amlodipine in order to treat his hypertension. [49]
Iron-deficiency anemia is mainly caused by blood loss, such as may occur during menses or gastrointestinal hemorrhage.This often results in a depapilled, atrophic glossitis, giving the tongue a bald and shiny appearance, along with pallor (paleness) of the lips and other mucous membranes a tendency towards recurrent oral ulceration, [6] and cheilosis (swelling of the lips). [7]
Here are the common causes of a metallic taste in mouth, and how you can get rid of it. Skip to main content. Sign in. Mail. 24/7 Help. For premium support please call: 800-290-4726 more ...
Iron metabolism disorders may involve a number of genes including HFE and TFR2. [1]Hepcidin is the master regulator of iron metabolism and, therefore, most genetic forms of iron overload can be thought of as relative hepcidin deficiency in one way or another.
Before iron deficiency anemia sets in, “the body will do everything it can to retain the right number of red blood cells. So, it will deplete the storage iron before it depletes the red blood ...