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An incentive spirometer is a handheld medical device used to help patients improve the functioning of their lungs. By training patients to take slow and deep breaths, this simplified spirometer facilitates lung expansion and strengthening. Patients inhale through a mouthpiece, which causes a piston inside the device to rise.
The Journal of Human Hypertension is a monthly peer-reviewed medical journal covering research into hypertension. It was established in 1987 and is published by Nature Publishing Group . The editor-in-chief is Michael Stowasser ( University of Queensland ).
Clinical and Experimental Hypertension is a peer-reviewed medical journal that covers all aspects of human and animal hypertension. It was established in 1978 and is published by Taylor & Francis . The editor-in-chief is Mustafa F. Lokhandwala ( University of Houston ).
Hutchinson published his paper about his water spirometer and the measurements he had taken from more than 4,000 subjects, [2] describing the direct relationship between vital capacity and height and the inverse relationship between vital capacity and age. He also showed that vital capacity does not relate to weight at any given height.
Spirometry (meaning the measuring of breath) is the most common of the pulmonary function tests (PFTs). It measures lung function, specifically the amount (volume) and/or speed (flow) of air that can be inhaled and exhaled.
The American Journal of Hypertension is a monthly peer-reviewed medical journal covering the field of cardiovascular medicine. It is published by Oxford University Press and the editor-in-chief is Ernesto L. Schiffrin (McGill University). According to the Journal Citation Reports, the journal has a 2022 impact factor of 3.2. [1]
Hypertension is a monthly peer-reviewed scientific journal [1] that was established in 1979. It is published on behalf of the American Heart Association by Lippincott Williams & Wilkins . The editor-in-chief is Rhian M. Touyz.
Lung volumes. Functional residual capacity (FRC) is the volume of air present in the lungs at the end of passive expiration. [1] At FRC, the opposing elastic recoil forces of the lungs and chest wall are in equilibrium and there is no exertion by the diaphragm or other respiratory muscles.