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Alfuzosin should not be used for patients with renal impairment. [40] [33] Tamsulosin is excreted via urine and 9% of that is unchanged on its active form, elimination half-life for tamsulosin is between 9–13 hours for healthy volunteers. The elimination half-life for target patients is around 14–15 hours.
Alfuzosin, sold under the brand name Uroxatral among others, is a medication of the α 1 blocker class. It is used to treat benign prostatic hyperplasia (BPH). [1]As an antagonist of the α 1-adrenergic receptor, it works by relaxing the muscles in the prostate and bladder neck, making urination easier.
Tamsulosin is a selective α 1 receptor antagonist that has preferential selectivity for the α 1A receptor in the prostate versus the α 1B receptor in the blood vessels. [ 25 ] When alpha 1 receptors in the bladder neck, prostate, ureter, and urethra are blocked, a relaxation in smooth muscle tissue results. [ 16 ]
How to Reset Female Hormones For Weight Loss. This article was reviewed by Craig Primack, MD, FACP, FAAP, MFOMA. Hormones can be helpful heroes, supporting the immune system and a healthy sleep ...
Alfuzosin [4] Doxazosin [5] Prazosin (inverse agonist) [6] Tamsulosin [7] Terazosin [8] Silodosin [9] Selective α 2-adrenergic receptor antagonists include: Atipamezole; Idazoxan; Mirtazapine; Yohimbine; Finally, the agents carvedilol and labetalol are both α and β-blockers. Below are some of the most common drugs used in the clinic.
Flu A and flu B are the most common strains of the flu that circulate in humans. The U.S. is currently in the middle of flu season, with a high number of cases reported across the country. There ...
A Washington, D.C. man has been charged with murder after police say he stabbed his grandmother to death and then texted a photograph of her dead body to other family members last Friday.
Alpha-1 blockers such as alfuzosin, doxazosin, tamsulosin, and silodosin involve CYP450 enzyme metabolism, particularly by CYP3A4. [14] Alpha-1 blockers will conjugate in glucuronidation during metabolism. CYP3A4 inhibitors inhibit glucuronidation and hence reduce the glucuronide-conjugated metabolite. [15]