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Causes may include heart failure, kidney failure, narcotic poisoning, intracranial pressure, and hypoperfusion of the brain (particularly of the respiratory center). The pathophysiology of Cheyne–Stokes breathing can be summarized as apnea leading to increased CO 2 which causes excessive compensatory hyperventilation, in turn causing decreased CO 2 which causes apnea, restarting the cycle.
Cheyne–Stokes respiration: John Cheyne, William Stokes: palliative care: respiratory center damage: fluctuation between apnoea and tachypnoea: Churchill–Cope reflex: Edward Delos Churchill, Oliver Cope: cardiology: heart failure: distension of pulmonary vascular bed causes tachypnoea: Chvostek sign: František Chvostek: endocrinology ...
Cheyne–Stokes respiration is a breathing pattern consisting of alternating periods of rapid and slow breathing, which may result from a brain stem injury. [12] Cheyne-Stokes respiration may be observed in newborn babies, but this is occasionally physiological (normal). Chest retractions may be observed in patients with asthma.
The FOUR Score assesses four domains of neurological function: eye responses, motor responses, brainstem reflexes, and breathing pattern. The rationale for the development of the FOUR Score constituted creation of a clinical grading scale for the assessment of patients with impaired level of consciousness that can be used in patients with or ...
Another common mechanism that causes CSA is the loss of the brain's wakefulness drive to breathe. [11] Screenshot of a PSG system showing a central apnea. CSA is organized into 6 individual syndromes: Cheyne-Stokes respiration, Complex sleep apnea, Primary CSA, High altitude periodic breathing, CSA from medication, CSA from comorbidity. [11]
An increase in pressure, most commonly due to head injury leading to intracranial hematoma or cerebral edema, can crush brain tissue, shift brain structures, contribute to hydrocephalus, cause brain herniation, and restrict blood supply to the brain. [13] It is a cause of reflex bradycardia. [14]
Both Cheyne–Stokes breathing (the alternation of apnoea with tachypnoea) and Stokes–Adams syndrome are named after him. Stokes' sign is a severe throbbing in the abdomen, at the right of the umbilicus, in acute enteritis. Stokes law is that a muscle situated above an inflamed membrane is often affected with paralysis.
Central neurogenic hyperventilation (CNH) is an abnormal pattern of breathing characterized by deep and rapid breaths at a rate of at least 25 breaths per minute. Increasing irregularity of this respiratory rate generally is a sign that the patient will enter into coma.