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Portal hypertensive gastropathy can also be treated with endoscopic treatment delivered through a fibre-optic camera into the stomach. Argon plasma coagulation and electrocautery have both been used to stop bleeding from ectatic vessels, and to attempt to obliterate the vessels, but have limited utility if the disease is diffuse.
The lamina propria is a thin layer of loose (areolar) connective tissue, which lies beneath the epithelium, and together with the epithelium and basement membrane constitutes the mucosa. As its Latin name indicates, it is a characteristic component of the mucosa, or the mucosa's "own special layer."
Underlying the epithelium is the lamina propria, which contains myofibroblasts, blood vessels, nerves, and several different immune cells, and the muscularis mucosa which is a layer of smooth muscle that aids in the action of continued peristalsis and catastalsis along the gut.
Reactive gastropathy is morphologically distinct entity [3] [4] that can be separated from gastritis, which by definition has a significant inflammatory component.. As a reactive gastropathy may mimic a (true) gastritis symptomatically and visually in an endoscopic examination, it may incorrectly be referred to as a gastritis.
Gastric erosion occurs when the mucous membrane lining the stomach becomes inflamed. Specifically, the term "erosion" in this context means damage that is limited to the mucosa, which consists of three distinct layers: the epithelium (in the case of a healthy stomach, this is non-ciliated simple columnar epithelium), the basement membrane, and the lamina propria.
Watermelon stomach, watermelon disease: Endoscopic image of gastric antral vascular ectasia seen as a radial pattern around the pylorus before (top) and after (bottom) treatment with argon plasma coagulation: Specialty: Gastroenterology Symptoms: Bleeding in the stomach and intestines, edema, dilated blood vessels
Histopathological features of this disease include fibrosis obliterating the lamina propria and smooth muscle fibers extending from a thickened muscularis mucosa to the lumen. [12] SRUS has been treated with a variety of methods, including conservative measures such as diet and bulking agents, medical therapy, biofeedback, and surgery.
In addition eotaxin has been shown to have an integral role in regulating the homing of eosinophils into the lamina propria of stomach and small intestine. [18] In the allergic subtype of disease, it is thought that food allergens cross the intestinal mucosa and trigger an inflammatory response that includes mast cell degranulation and ...