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Paroxysmal sympathetic hyperactivity (PSH) is a syndrome that causes episodes of increased activity of the sympathetic nervous system. Hyperactivity of the sympathetic nervous system can manifest as increased heart rate, increased respiration, increased blood pressure, diaphoresis , and hyperthermia . [ 1 ]
The underlying mechanism involves the nervous system slowing the heart rate and dilating blood vessels, resulting in low blood pressure and thus not enough blood flow to the brain. [2] Diagnosis is based on the symptoms after ruling out other possible causes. [3] Recovery from a reflex syncope episode happens without specific treatment. [2]
Studies with 8-Oxo-2'-deoxyguanosine varied by methods of measurement and type of depression, but F2-isoprostane level was consistent across depression types. Authors suggested lifestyle factors, dysregulation of the HPA axis, immune system and autonomics nervous system as possible causes. [155]
Hyperreflexia is overactive or overresponsive bodily reflexes. Examples of this include twitching and spastic tendencies, which indicate disease of the upper motor neurons and the lessening or loss of control ordinarily exerted by higher brain centers of lower neural pathways. [citation needed] Spinal cord injury is the most common cause of ...
Sympathicotonia is a stimulated condition of the sympathetic nervous system, marked by vascular spasm elevated blood pressure, and goose bumps. [26] [27] Heightened sympathetic nervous system activity is also linked to various mental health disorders such as, anxiety disorders and post-traumatic stress disorder (PTSD). It is suggested that the ...
These two subunits are the sympathetic nervous system and the parasympathetic nervous system. The sympathetic response is colloquially known as the "fight-or-flight response," indicated by accelerated pulse and respiration rates, pupil dilation, and a general feeling of anxiety and hyper-awareness. This is caused by the release of epinephrine ...
Overactive PKC signalling may lead to long term structural changes in the frontal cortex as well, potentially leading to progression of manic symptoms. [ 102 ] [ 103 ] Glycogen synthase kinase 3 has been implicated in bipolar disorder, as bipolar medications lithium and valproate have been shown to increase its phosphorylation, thereby ...
Mixed anxiety–depressive disorder should only be considered as a diagnosis when the symptoms impede a person's functioning in day-to-day life and/or decrease their quality of life and symptoms of anxiety and depression are roughly in equal measure without the severity of major depressive disorder or an anxiety disorder.
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