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Corneal opacification is a term used when the human cornea loses its transparency. The term corneal opacity is used particularly for the loss of transparency of cornea due to scarring. Transparency of the cornea is dependent on the uniform diameter and the regular spacing and arrangement of the collagen fibrils within the stroma.
Contact lens-induced hypoxia triggers the cornea to undergo anaerobic respiration, resulting in a buildup of lactic acid that osmotically draws water into corneal cells, causing edema. [6] Two explanations have been proposed for contact lens-induced stromal thinning. It is thought that this edema may inhibit stromal tissue synthesis. [4]
The Chandler variant of ICE is characterized by pathology on the inner surface of the cornea leading to abnormal endothelial pump function. [2] [5] Other features include possible mild iris changes, corneal edema, and normal to slight elevations in intraocular pressure. [1] [5] Cogan-Reese variant is characterized by multiple pigmented iris ...
Recurrent corneal erosion (RCE) is a disorder of the eyes characterized by the failure of the cornea's outermost layer of epithelial cells to attach to the underlying basement membrane (Bowman's layer). The condition is excruciatingly painful because the loss of these cells results in the exposure of sensitive corneal nerves.
Corneal hydrops might be caused by a tear in the recently discovered Dua's layer, a 15 micron thick layer between the corneal stroma and Descemet's membrane, Harminder Dua suggests that this finding will affect corneal surgery, including penetrating keratoplasty, and understanding of corneal dystrophies and pathologies, such as acute hydrops. [5]
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Corneal abnormalities including edema or opacities ("corneal haze") Corneal staining; Pupil abormalities including abormal pupil size; Abnormal intraocular pressure; Severe pain; The most useful is a smaller pupil in the red eye than the non-red eye (opposite eye) and sensitivity to bright light. [2]
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