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Up to 17% of cats with HCM experience arterial thromboembolism, but cats with other cardiomyopathies are also at increased risk. Cats with abnormally increased hemostasis, which can occur with hyperthyroidism, tumors, extensive inflammation, blood poisoning (sepsis), injury, or disseminated intravascular coagulation, represent another risk ...
Feline hypertrophic cardiomyopathy (HCM) is the most common heart disease in domestic cats; [69] [70] [71] the disease process and genetics are believed to be similar to the disease in humans. [72] In Maine Coon cats, HCM has been confirmed as an autosomal dominant inherited trait. [73] Numerous cat breeds have HCM as a problem in the breed. [74]
In cats, taurine deficiency is the most common cause of dilated cardiomyopathy. [47] As opposed to these hereditary forms, non-hereditary DCM used to be common in the overall cat population before the addition of taurine to commercial cat food.
Not all cats that test positive will have clinical signs of the disease, and some Maine Coon cats with clinical evidence of hypertrophic cardiomyopathy test negative for this mutation, strongly suggesting that a second mutation exists in the breed. The HCM prevalence was found to be 10.1% (95% CI 5.8 -14.3%) in this study. [39]
Feline disease refers to infections or illnesses that affect cats. They may cause symptoms, sickness or the death of the animal. Some diseases are symptomatic in one cat but asymptomatic in others. Feline diseases are often opportunistic and tend to be more serious in cats that already have concurrent sicknesses.
The Sphynx cat has a high rate of heart disease, either as HCM or mitral valve dysplasia. In a 2012 study of 114 Sphynx cats, 34% were found to have an abnormal heart, with 16 cats having mitral valve dysplasia and 23 cats having HCM. [27] These prevalences were found in cats with an average age of 2.62 years.
Cats lack the necessary glucuronyl transferase enzymes to safely break paracetamol down and minute portions of a normal tablet for humans may prove fatal. [24] Initial symptoms include vomiting, salivation and discolouration of the tongue and gums. After around two days, liver damage is evident, typically giving rise to jaundice.
Prognosis varies depending on the stage of the disease, with both a high recovery and mortality rate at different stages. The disease is reversible through intense feeding. [ 3 ] Treatment may involve the insertion of a temporary feeding tube to ensure adequate caloric intake for cats that have stopped eating as a result of this disease.