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Human papillomavirus infection (HPV infection) is caused by a DNA virus from the Papillomaviridae family. [5] Many HPV infections cause no symptoms and 90% resolve spontaneously within two years. [1] In some cases, an HPV infection persists and results in either warts or precancerous lesions. [2]
Papillomaviridae is a family of non-enveloped DNA viruses whose members are known as papillomaviruses. [1] Several hundred species of papillomaviruses, traditionally referred to as "types", [2] have been identified infecting all carefully inspected mammals, [2] but also other vertebrates such as birds, snakes, turtles and fish.
HPV is spread by direct and indirect contact from an infected host. Avoiding direct contact with contaminated surfaces such as communal changing rooms and shower floors and benches, avoiding sharing of shoes and socks and avoiding contact with warts on other parts of the body and on the bodies of others may help reduce the spread of infection.
The infection first appears as a red and swollen area on the skin, followed by development of circular papilloma warts and keratinized horny warts. Although transmission between rabbits is high, the tumors themselves do not contain the infectious virus. 25% of Papilloma infections become malignant and form squamous cell carcinoma. Metastases ...
HPV oral infection precedes the development of HPV+OPC. [8] [5] Slight injuries in the mucous membrane serve as an entry gate for HPV, which thus works into the basal layer of the epithelium. [14] [15] People testing positive for HPV type 16 virus (HPV16) oral infection have a 14 times increased risk of developing HPV+OPC. [14]
Gardasil is designed to prevent infection with HPV types 16, 18, 6, and 11. HPV types 16 and 18 currently cause about 70% of cervical cancer cases, [13] [14] and also cause some vulvar, vaginal, [11] penile and anal cancers. [12] HPV types 6 and 11 are responsible for 90% of documented cases of genital warts. [17]
In November, at the International Papillomavirus Conference, we presented new clinical and real-world data for Gardasil 9 that examined the prevalence of oral HPV infection, the burden of HPV ...
Genital HPV infections have an estimated prevalence in the US of 10–20% and clinical manifestations in 1% of the sexually active adult population. [29] US incidence of HPV infection has increased between 1975 and 2006. [29] About 80% of those infected are between the ages of 17 and 33. [29]